Sergei Funikov | Researcher | PhD | Engelhardt Institute of…

Sergei Funikov, Researcher | Cited by 425 | of Engelhardt Institute of Molecular Biology, Moscow (EIMB) | Read 76 publications | Contact Sergei Funikov
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Publications - CrestOptics

Images were acquired with X-Light V3.
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Retrotransposon activation contributes to neurodegeneration in…

Author summary Functional abnormality of TAR DNA-binding protein 43 (TDP-43), an aggregation-prone RNA and DNA binding protein, is observed in the vast majority of both familial and sporadic ALS cases and in ~40% of FTLD cases, and…
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Děčín - Wikipedia

Děčín (germană Tetschen-Bodenbach) este un oraș cu 51.820 de locuitori situat pe Elba, în regiunea Ústí nad Labem din nordul Cehiei, la granița cu Germania.
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Stress Granule Assembly Is Mediated by Prion-like Aggregation…

TIA-1 is an RNA binding protein that promotes the assembly of stress granules (SGs), discrete cytoplasmic inclusions into which stalled translation initiation complexes are dynamically recruited in.
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Publications | Swiss National Center of Competence in Research…

Establishing a Swiss RNA research focus that consolidates and reinforces the position of Switzerland in RNA biology by a coordinated, interdisciplinary research program.
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CircRNA Array | Superior Circular RNA Expression Profiling to…

Arraystar Circular RNA Microarray, covering 13,617 circRNAs, is the first and only commercially available technology for sensitive and reliable expression profiling . CircRNA arrays have been cited by scientific publications.
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MicroRNA–Mediated Repression of the Seed Matur…

Fig. 4. Reduced Accumulation of miRNAs in Various miRNA Biogenesis Mutant Backgrounds as Determined by Northern Blotting.
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RNA Collaborative Seminar Series

RNA Collaborative seminars are held virtually every other Wednesday, from 4:00-5:00 pm EDT (8:00 -9:00 pm GMT) and are recorded and made freely available for viewing on the RNA Collaborative YouTube Channel.
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Is imprinting the result of “friendly fire” by the…

In 1993, Denise Barlow proposed that genomic imprinting might have arisen from a host defense mechanism designed to inactivate retrotransposons.
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